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   p53基因 在 泌尿科学 分类中 的翻译结果: 查询用时:1.236秒
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p基因     
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  p 53 gene
    Effect of Expression of p53 Gene Driven by hTERT Gene Promoter on T24 Apoptosis
    hTERT启动子调控p53基因表达对T24细胞凋亡的作用
短句来源
    p53 GENE MUTATION IN HUMAN BLADDER CANCER
    p53基因在人膀胱肿瘤中的突变
短句来源
    Transfection of wild-type p53 gene and growth of bladder cancer cells
    野生型p53基因导入对膀胱癌细胞生长抑制和H-ras基因表达调控
短句来源
    Abnormal expression and clinical significance of muta-tion p53 gene on human bladder cancer
    突变型p53基因在人膀胱肿瘤中的表达及临床意义
短句来源
    Transforming growth factor αand P53 gene expression in bladder cancer
    膀胱癌转化生长因子α和P53基因表达及生物学意义
短句来源
更多       
  p 53
    Effect of Expression of p53 Gene Driven by hTERT Gene Promoter on T24 Apoptosis
    hTERT启动子调控p53基因表达对T24细胞凋亡的作用
短句来源
    p53 GENE MUTATION IN HUMAN BLADDER CANCER
    p53基因在人膀胱肿瘤中的突变
短句来源
    Expression of p53 Protein in Transitional Cell Carcinoma of the Bladder and It's Clinical Significance
    膀胱癌细胞p53基因蛋白表达的临床意义
短句来源
    Transfection of wild-type p53 gene and growth of bladder cancer cells
    野生型p53基因导入对膀胱癌细胞生长抑制和H-ras基因表达调控
短句来源
    Abnormal expression and clinical significance of muta-tion p53 gene on human bladder cancer
    突变型p53基因在人膀胱肿瘤中的表达及临床意义
短句来源
更多       
  p 53 genes
    Expression and interaciton of P21WAF_1/CIP_1 and P53 genes in human bladder carcinoma and the clinical significance
    P21WA F_1/CIP_1和P53基因表达与膀胱癌生物学行为的关系
短句来源
    Deletion of WTl and P53 genes in Wilms' tumor tissues
    肾母细胞瘤组织WT1、P53基因缺失检测
短句来源
    (4) Four cases of BTCC exhibited mutation of both ras and p53 genes.
    4例膀胱移行细胞癌存在ras基因及p53基因共同突变。
短句来源
    Conclusions High T G Fαm R N Aexpression might be an early eventin the develop ment of bladder cancer while abnorm al expressionof E G F Rand p53 genes might be related to the differentiation and malignancy of bladder cancer .
    结论  T G Fαm R N A 高表达可能是膀胱癌发生的早期事件。 E G F R 和p53 基因异常表达可能与膀胱癌的分化和恶性度相关。
短句来源
    Methods: WT1 and P53 genes in 30 sporadic Wilms' tumor tissues (6 epithelium predominated Wilms' tumor, 14 metrocyte predominated Wilms', and 10 stroma predominated Wilms' tumor) were studied by southern bolt hybridization with WT1 cDNA and P53 cDNA probes.
    方法:30例散发性肾母细胞瘤组织,其中上皮优势型6例,母细胞优势型14例,间质优势型10例,采用Southern印迹分子杂交技术检测肾母细胞瘤组织WT1、P53基因缺失,并分析其与组织类型的关系。
短句来源
更多       
  p 5 3 gene
    Effect of Expression of p53 Gene Driven by hTERT Gene Promoter on T24 Apoptosis
    hTERT启动子调控p53基因表达对T24细胞凋亡的作用
短句来源
    p53 GENE MUTATION IN HUMAN BLADDER CANCER
    p53基因在人膀胱肿瘤中的突变
短句来源
    Transfection of wild-type p53 gene and growth of bladder cancer cells
    野生型p53基因导入对膀胱癌细胞生长抑制和H-ras基因表达调控
短句来源
    Abnormal expression and clinical significance of muta-tion p53 gene on human bladder cancer
    突变型p53基因在人膀胱肿瘤中的表达及临床意义
短句来源
    Transforming growth factor αand P53 gene expression in bladder cancer
    膀胱癌转化生长因子α和P53基因表达及生物学意义
短句来源
更多       

 

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      p 53 gene
    The p53 Gene Family: Control of Cell Proliferation and Developmental Programs
          
    Loss of function of the p53 gene occurs in virtually every case of cancer, and deficiency in p53 is an unavoidable prerequisite to the development of malignancies.
          
    This review is focused on strategies of the p53 gene, demonstrating individual mechanisms underlying its functions.
          
    New mutations in the human p53 gene - a regulator of the cell cycle and carcinogenesis
          
    We studied p53 gene mutations in 26 primary tumors of colorectal cancer patients.
          
    更多          
      p 53
    Further, manzamine A (50 μM) substantially down regulated the expression of p53 while sarcophine (50 μM) slightly induced the level of p21.
          
    The aim of this study was to investigate the role of pirh2 (p53-induced RING-H2) protein in the proliferation, apoptosis and cell cycle control of the lung cancer cell line A549.
          
    However pathway involved in such signaling at least partially is independent of p53 function.
          
    Such are the suggested effects on telomerase of Myc, p53, Waf1, protein kinases B and C, Wnt5A, TGFβ, WT1, and estrogens.
          
    However, Myc, p53, WT1, estrogens, protein kinases B and C, and TGFβ can also directly influence telomerase independently of the G1-S checkpoint mechanism.
          
    更多          
      p 53 genes
    The transcriptional levels of the Bax and p53 genes increased significantly in U2OS cells transfected with pcDNA3.1(+)/azurin, but the Bcl-2 mRNA level decreased.
          
    This is closely associated with the up-regulation of the transcriptional level of the Bax and p53 genes, and the down-regulation of that of the Bcl-2 gene.
          
    These results indicated that activated C-erbB-2, N-ras and altered p53 genes may have a role in human HCC pathogenesis through promiting the development of HCC from hepatic cirrhosis and the progression of HCC.
          
    Several circulating colorectal cancer DNAs have been identified by PCR detection such as the activated K-ras oncogene, inactivated APC, p53 genes, and microsatellite instability (MSI).
          
    Using a microsatellite analysis technique, we analyzed this area and regions in the vicinity of the APC, DCC, and p53 genes.
          
    更多          


    OBJECTIVE To investigate the effects of Chinese herb astragalus membranaceus(AM)on mRNA expressions of hypothalamic vasopressin(AVP),renal AVP V2 receptor,and AVP dependent AQP2 in rats with adriamycin induced nephrotic syndrome. METHODOLOGY Male SD rats were randomly grouped into untreated adriamycin induced nephrotic group(NS group),AM treated adriamycin induced nephrotic group(NS+AM group)and normal control group(NC group).Rats in NS+AM group received...

    OBJECTIVE To investigate the effects of Chinese herb astragalus membranaceus(AM)on mRNA expressions of hypothalamic vasopressin(AVP),renal AVP V2 receptor,and AVP dependent AQP2 in rats with adriamycin induced nephrotic syndrome. METHODOLOGY Male SD rats were randomly grouped into untreated adriamycin induced nephrotic group(NS group),AM treated adriamycin induced nephrotic group(NS+AM group)and normal control group(NC group).Rats in NS+AM group received 0 5g/(kg·day) of AM peritoneally for 4 weeks before sacrifice,while rats in other groups received normal solution in a same way.Hypothalamic AVP mRNA expression was examined by dot hybridization assay.RT PCR was applied for detection of the renal mRNA expression of V2 receptor and AQP2.The readings of the RT PCR were normalized by GAPDH mRNA in the same sample. RESULTS All rats receiving adriamycin presented typical nephrosis.No significant difference in plasma osmolarity was detected among NS,NS+AM and NC rats.Hypothalamic AVP mRNA expression was higher in NS rats than in NC rats(53 59±5 49 vs .25 72±1 96, P <0 01).AM completely reversed this up regulated expression(21 88±1 25).Both in cortex and medulla of the kidney,AVP V2 expression was found increased by 169% and 55% respectively in nephrotic rats,as compared to those in NC rats.The increment of AQP2 mRNA expression in NS rats was in consistence with that of V2 receptor.AM partially reversed the up regulated expression of V2 and AQP2 mRNA. CONCLUSION The up regulated mRNA expressions of hypothalamic AVP,renal V2 receptor and AQP 2 might play a role in edema formation in adriamycin induced nephrotic rats.AM exerts its therapeutic effects on nephrosis partially through this mechanism.

    目的:研究下丘脑精氨酸血管加压素( A V P) 和其 V2 受体与 A V P 依赖性水通道水孔蛋白2( A Q P2 )在阿霉素肾病综合征大鼠肾脏表达的情况,及中药黄芪对此的影响。 方法:下丘脑 A V P m R N A 检测用斑点杂交法,肾脏皮髓质 A V P V2 受体、 A Q P2 m R N A 检测采用半定量 R T P C R 法。 结果:肾病大鼠下丘脑 A V Pm R N A表达较正常对照增强( A:5359 ±549 vs 2572 ±196 , P< 001) ,黄芪可纠正此种高表达( A:2188 ±125) 。半定量 R T P C R 检查发现肾脏皮质和髓质 V2 受体、 A Q P2 m R N A 表达均呈现有意义的上调,黄芪则可使这些变化缓解( P 均< 001) 。 结论:在阿霉素肾病大鼠中存在下丘脑 A V P 基因表达的增加,肾脏 A V P V2 受体和 A Q P2m R N A 表达的上调,这些变化可能是肾病水潴留的机制之一,中药黄芪能改善这些变化。

     
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